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A Rapid Review of VIVA on Nervous System



By Waseem Shehzad (PMC)
waseem_dr@hotmail.com

Q.NO.1#

SUMMARY OF NEUROLOGICAL HISTORY TAKING

1.     PRESENTING COMPLAINTS

    • Time relationships
    • Localization
    • Trigger factors
    • Associated features
  1. PAST MEDICAL HISTORY

·         Birth / pregnancy

·         Head and spine injury

·         Infections (meningitis, encephalitis)

·         Surgical procedures

·         Drug therapy

3.     FAMILY HISTORY

·         Epilepsy, migraine, multiple sclerosis, stroke, cerebral aneurism,muscle disorders, dementia, spinocerebellar degenerations, and neuropathies

4.     SOCIAL HISTORY

·         Occupation

·         Marital status

·         Smoking habit

·         Alcohol consumption

·         Recreational drugs

·         Sexual orientation and habits

Q.NO.2. COMMON CAUSES OF HEADACHE

1.    EXTRACRANIAL DISORDERS

§         Temporomendibular joint(osteoarthritis)

§         Cervical spine(cervical spondilosis)

§         Opthalmological(glaucoma)

§         Teeth(dental sepsis_)

§         Middle ear(earache)

§         Paranasal sinuses(sinusitis)

Arteries(cranial arthritis)

2.    INTRACRANIAL DISORDERS

§     Migraine

§     Meningitis

§     Intracranial hypertension

§     Tumour

§     Subarachnoid bleed

3.    PSYCHOMOTOR DISORDERS

§     Tension headache

QUESTION . NO. 3

CLASSIFICATION OF SEIZURES

          GENERALIZED SEIZ.

§     Absence attacks--petit mal (3 hz spike and wave)

§     Myoclonic seizures--myoclonic jerks

§     Tonic---clonic seizures of grand mal or major fits

§     Tonic seizures

§     Atonic or akinetic seizures

              PARTIAL SEIZURES

§     Simple partial seiz.—no loss of conciousness

§     Complex partial seiz.--------loss of conciousness

§     Partial seiz. Evolving into generalized seiz.

§     Generalized seiz. With only EEG evidence  of local onset

PSEDO SEIZURES-----------no epileptic attacks

Q . NO. 4#

CHECKLIST FOR HISTORY OF FALLS

WAS IT AN ACCIDENTAL FALL.?

§    Recall of event

§    Trip or slip

§    Walking surface

§    Footware

§    Illumination

§    Visual acuity

WAS IT A SPONTANEOUS FALL?

§    Preceding symptoms

§    Onset with head movement or standing upright

§    Ass. Symptoms pf vertigo deafness or tinnitus

§    Ass. Chest pain or palpitation

§    Awareness of hitting the ground

§    Eyewitness account

§    Resulting injuries or incontinence

§    Recall of events att  the time

§    Ability to regain erect posture

§    Previous confusion , dementia. Or parkinsonism

§    Previous hypertension, blackout, or epilepsy

§    Dryg therapy----antidepressants, hypnotics, hypotensive agents,

§    Alcohol consumption

Q. NO. 5#

THE NERVOUS SYSTEM-GENERAL OBSERVATIONS

§   Gait

§   Rombergs, test

§   Speech and handwriting

§   Mental status

§   Facial appearance

§   Involuntary movements

§   Movements of neck and soine

Q. NO 6#

CAUSES OD DYSARTHRIA AND DYSTONIA

MECHANISM

EXAMPLES

Weakness of facial and tongue muscles

Myasthenia gravis

Lesions of lower brain stem

Motor neuron disease

Bilateral corticosp. Tract lesion above the pons

Multiple lacunar infarcts

Impaired control of phonation and  articulation

Parkinsonism

Imprecise motor control system

Cerebellar lesions

Impaired larynx function

Rec. laryngeal . n palsy

Q.NO.7#

EXAMINTION OF SPEECH AND LANGUAGE

SPONTANEOUS SPEECH

Naming objects and concepts

Articulation

Comprehension of spoken language

Fluency

Repetition of spoken phrases

Paraphasias

Reading aloud

Grammer

Handwriting

Syntax

 

Q. NO.8#

ETIOLOGICAL CLUES IN THE HISTORY OF EPILEPSY

FEATURES

POSSIBLE CAUSE

Childhood onset

Idiopathic

Headache

Intracerebral bleed or  tumour

Focal symptoms

Intracerebral tumour

Nocturnal fits

Focal lesions and hypoglycemia

Alcohol abuse

Alcohol withdrawal

Subdural hematoma

Hypoglycemia

Sexual activity or drug abuse

Hiv infection

Drug therapy

Iatrogenic

Pregnancy

Ecclampsia

Hypertension/CVA

Cerebrovascular disease

Q.NO.9#

PATTERNS OF VERTIGO AND THEIR CAUSES

ACUTE

Viral vestibular neuronitis

Skull fracture

Multiple sclerosis

Meniere,s disease

ACUTE AND RECURRENT

Benign positional vertigo

Meinere,s disease

Migraine

Chronic otitis media

Multiple sclerosis

Acoustic neuroma

CHRONIC AND PERSISTENT

Multiple sclerosis

Ototoxic drugs

Vertibrobasilar ischemia

Acoustic neuroma

Q NO 10.#

COMMON CAUSES OF HEADACHE

Extracranial disorders

Example

Temporomendibular joint

Osteoarthritis

Cervical spine

Cervical spondilosis

Opthalmological

Glaucoma

Teeth

DENTAL SEPSIS

Middle ear

Earache

Paranasal sinuses

Sinusitis

Arteries

Cranial arteritis

INTRACRANIAL DISORDERS

§        Migraine

§        Meningitis

§        Intracranial hypertension

§        Tumour

§        Subarachnoid bleed

PSYCHOMOTOR  DISORDERS

§        Tension headache

Q.NO.11#

RELATION B/W CAUSE AND SITE OF HEADACHE

DISORDER

SITE

Subarach. Hemorrhage

Bilateral generalized or nuchal

Tension headache

Same

Migraine

Unilateral, diff sides on separate occasions

Cluster headache

Unilat, eye ,nose,cheek

Cranial arteritis

Unilat. Wth scalp tenderness

Trigeminal neuralgia

Unilat. Maxillary or mandibular branch of trigeminal nerve

Post hepatic neuralgia

Unilat. Ophthalmic br of trigeminal nerve

Q.NO12#

DRUGS AFFECTING THE PUPIL

DILATED OR MYDRIASIS

MODE OF ACTION

Atropine/ homatropine

Anticholinergic

Amphetamine and derivatives

Sympathomometics

CONSTRICTED OR  MIOSIS

 

Neostigmine, morphine and derivatives

parasympathomimetics

COMMON FEATURES IN OPTIC NERVE LESIONS

ABNORMALITIES

COMMON CAUSES TO CONSIDER

Abn. Papillary response

Optic neuritis /multiple sclerosis

Enlargement of blind spot

Vascular disease

Scotomas

Retrobulbar and paraseller neoplasms e.g pituitary adenomas

Impaired visual acuity

 

Impaired color vision

Cranoipharangioma and meningioma

Abnormal fundoscopy

 

Q. NO 13#

COMMON CAUSES OF VII NERVE PALSIES

UNILATERAL

BILATERAL

 

UPPER MOTOR NEURON TYPE WEAKNESS

Usually vascular

Often vascular

Cerebral tumour

Consider motor neuron disease

Multiple sclerosis

 

LOWER MOTOR NEURON TYPE WEAKNESS

Usually BELLS palsy

Neurosarcoidosis

Consider parotid tumour

 

Myasthenia gravis

Head injuries

Generalized polyneuropathies

Skull base tumours

Guillain barre syndrome

Some myopathies e.g myotonic dystrophy

Q.NO 14#

COMMON CAUSES OF DEAFNESS

Conduction Deafness

Wax I the external canal

Damage to tympanic memb

Fluid in inner ear

Ossicular chain disruption

Otosclerosis

Diminished  hearing on the affected side

Rinne,s test ;bone conduction louder’

Weber,s test ; to the affectd side

 

SENSORINEURAL DEAFNESS

EFFECT

Damage to cochlear nerve and organ of corti

Acoustic neuroma’

Transverse fracture of petrous temporal bone

Bilaterally in the elderly

(degenerative)

Diminished hearing on the affected side

Rinne,s test ;air conduction louder

Weber,s test;to unaffected side

Q.NO 15#

COMMON CAUSES OF IX AND X NERVE LSESIONS

UNILATERAL OF IX AND X

§        Skull base neoplasms including meningioma

§        Skull base factures

RECURRENT LARYNGEAL

§       Bronchial carcinoma   

§       Mediastinal lymphoma

§       Aortic aneurism

§       BRONCHIAL carcinoma

§       Mediastinal lymphoma

§       Aortic arch aneurism

BILATERAL X

§       Progressive bulbar palsy

§       Bilateral supranuclear lesions

§       Cerbrovascular disease

§       Multiple sclerosis

Q. NO 16#

PARALYSIS OF MOTOR DYSFUCTION

§       Paralysis or weakness

§        Impairment of coordination

§        Changes in tone and posture –dystonia

§        Involuntary movements---dyskinesia

§        Changes in the rate at wch movements are performed—hypokinesia and bradykinesia

§        Loss of learned movement pattern

Q. NO 17#

CAUSES OF MUCLE WEAKNESS

ANATOMICAL ETIOLOGY

ASS, FEATURES

COMMON CAUSES

LOWER MOTOR NEURON

Muscle atrophy

Fasciculations

Reklexes absent or diminished

Hypotonia

Peripheral neuropathy

Rediculopathies

Ant. Horn cell damage

e.g poliomyelitis

motor neuron disease

UPPER MOTOR NEURON

Patterned weakness

Little or no muscle wasting

Hypereflexia

Hypertonia

Hypokinesia of movement

Cerebrovascular diseaseas hemiplegia

Spinal injuy or disease

e.g paraplegia

multiple sclerosis

MYOPATHIES

Muscle wasting usually proximal

Hypotonias

Tenderness(myositis)

Heriditory conditions as muscular dystrophy

Alcohol and other toxins

PSYCHOLOGICAL

Inconsistent weakness

No ass. Features

Stress

Anxiety

Compensation claims

Q. NO 18#

TYPES PF TREMOR

NAME

FREWQUENCY

EXAMPLE

REST

POSTURE

Action  or postural

10

Hyperthy Anxiety and fatigue roidism

-

+

Intension

5

Cerebellar

-

+

Resting

5

Parkinsonism

++

+-

Q/NO.19#

MRC SCALE FOR MUSCLE POWER

0

NO muscle contraction visible

2

Joint movement when effect of gravity eliminated

1

1muscle cont visible but no  movement of joint

3

Movement sufficient to overcome the effect of gravity

4

Movement overcomes gravity and added resistance

5

Normal power

Q. NO 20#

ONE METHOD OF TESTING MOTOR SYSTEM

1)TEST IN A PROXIMO DISTAL DIRECTION

IN UPPER LIMB EXAM FOR

§      Abd. And add. Of shoulder

§      Flex. And ext. of shoulder

§      Flex. And ext. of elbow

§      Flex. And ext. of wrist

§      Sup. And pron. Of forearm

§      Extension of fingers at both MP AND IP joints

§      test abdominal muscles  BY ASKING THE SUPINE pt to flex neck or sit forward

IN LOWER LIMB EXAM. FOR

§     Hip flex. And ext. add. And abd.

§     Knee flex. And ext.

§    Foot dorsiflex. Planter flexion , inv. And eversion  , toe planter flex. And dorsiflexion

Q. NO 21#

SIGNS ATTRIBUTABLE TO LESIONS IN DIFF. PARTS AND PATHS OF NERVOUS SYSTEM

1)UPPER MOTOR NEURON LESIONS

§        Weakness and paralysis of movement

§        Inc. in tone of clasp knife type

§        Inc. amplitude o ftendon  reflexes

§        An extensor planter response

2)LOWER MOTOR NEURON LESIONS

§        Weakness and paralysis of muscles

§        Wasting of muscles and fasciculations

§        Red. In tone

§        Loss of tendon reflexes

 

3)EXTRAPYRAMIDAL LESIONS

·         Resting tremor

·         Rigidity cogwheel or lead pipe

·       Bradykinesia ,expressionless face, festinent gait

i)CEREBELLAR LESIONS

·         Ataxia

·         Intension tremor of limbs

·         Jerking nystagmus

·         Dysarthria of staccato or scanning type

·         Dysmetria and past pointing

·         Hypotonia and pendular  tendon reflexes

·         Smooth movements may be replaced wth jrking movements

1)GEN. NEUROPATHIES

·         Dim. Of superf. Sensation affecting the distal aspect of limbs  wth stocking and gloves distribution

·         Wasting and weakness of distal limb musculature

·         Early loss of tendonreflexes

2)SENSORY TRACTS

DORSAL COLOUMNS

·         ataxia of gait and limb movement aggravated by eye closure  +ve roomberg test

·         impaired position sense

·         dim. Appreciation of vibration

1.      LAT. SP. THALMIC TRACT

·         Impaiment of pain and temperature sense

2.      MUSCLES

·         Wasting and weakness usually proximal

·         Red. In reflexes when muscle wasting is marked

3.      CEREBRAL CORTEX DYSFUNCTION

·         Dysphasia and dyscalculia

·         Rt and Lt disorientation

·         Astereognosis sensory attention

·         Apraxia

·         Amnesia and cognitive disorder

·         Visual field homonymous defects

·         Hemiparesis , monoparesis

ABNORMALITIES OF INTENSITY OF IST HEART SOUND

 QUIET

§        Low cardiac output

§        Poor left ventricular function

§        Long P R interval

§        Ist degree heart block

§       Rheumatic mitral regurgitation

LOUD

§     inc cardiac output

§     Large st. vol

§     Mitral stenosis

§     Short P R interval

§     Atrial myxoma

§     VARIABLE

§     Atrial fibrillation

§     Extrasystoles

§     Comp. heart block

ABNORMALITIES OF SECOND HEART SOUND

QUIET

§  Low card. Output

§  Calcific aortic stenosis

§  Aortic incompetent

   LOUD

§Systemic hypertension

§Pulm. Hypertension

§SPLIT

§Widens in inspiration

§RBBB

§Pulm. Stenosis

§Pulm. Hypertension

§VSD

§Fixed splitting

§ASD

§Widens in expiration

§Aortic stenosis

§Hypertrophic cardiomyopathy

§LBBB

§Vent. Pacemaker

---------------------------------------------------------------------------

CAUSES OF 3RD HEART SOUND

1)PHYSIOLOGICAL

§         healthy young adults

§         athletes

§         pregnancy

§         fever

2)PATHOLOGICAL

§         large poor contracting  left ventricle

§         mitral reflux

GRADES OF INTENSITY  OF MURMUR

§       heard by an expert in optimum conditions

§       same by  a non expert

§       easily heard , no thrill

§       a loud murmur wth a thrill

§       very loud often heard over a large area  with a thrill

§       extremely loud heard with a steth.

 

CAUSES OF A SYSTOLIC MURMUR

EJECTION SYSTOLIC MURMUR

§       Inc. flow through normal valves

§       Innocent systolic murmur

§       Fever

§       Athletes

§       Pregnancy

§       ASD

NORMAL OR REDUCED FLOW THROUGH STENOTIC VALVE

§       Aortic and pulm. Stenosis

OTHER CAUSES OF FLOW MURMUR

§       Hypertrophic obst. Cardiomyopathy

§       Acute regurgitation

PANSYSTOLIC MURMUR

§       All caused by a systolic leak from a high  to a low pressure chamber

§       Mitral reg.

§       Tricuspid reg.

§       VSD

§       Leaking tricuspid or mitral prosthesis

§       Mitral valve prolapse

 

COMMON VENOUS ABNORMALITIES

DVT

§      varicose veins

§      superficial thrombophlebitis

§      chronic venous insufficiency  and ulceration

 

 

DEEP  VENOUS THROMBOSIS

Ask about

§     recent bed rest or operation

§     recent travel esp. long air travel

§     previous trauma to leg , esp . long bones fractures, plaster of paris splintage,and immobilization

§     pregnancy

§     previous DVT

§     family history of thrombosis

§     recent central venous cathetrisation  injections of  drugs

 

 

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